Nigel Emptage - Selected Publications#


Vasquez-Lopez SA, Turcotte R, Koren V, Ploschner M, Cizmar T, Padamsey Z, Booth MJ, Cizmar T, Emptage NJ (2018) Subcellular spatial resolution achieved for deep-brain imaging in vivo using a minimally invasive multimode fibre. Light: Science and Applications 7:110

Foster W, Padamsey Z, Galione A. Emptage NJ (2018) Hippocampal mGluR1-dependent long-term potentiation requires NAADP-mediated acidic store Ca2+ signaling. Science Signaling 11: eaat9093

Haslehurst P, Yang Z, Dholakia K, Emptage NJ (2018) Fast volume-scanning light sheet microscopy reveals transient neuronal events. Biomedical Optics Express 9 2154-2167.

Padamsey Z, Tong R, Emptage NJ (2017) Glutamate is required for depression but not potentiation of long-term presynaptic function. eLife 6, e29688. DOI: https://doi.org/10.7554/eLife.29688.

Padamsey Z, McGuinness L, Bardo SJ, Reinhart M, Tong R, Hedegaard A, Hart ML, Emptage NJ (2017) Activity-dependent exocytosis of lysosomes regulates the structural plasticity of dendritic spines. Neuron 93, 132-146.

Jeans AF, van Heusden FC, Al-Mubarak B, Padamsey Z, Emptage NJ (2017) Homeostatic presynaptic plasticity is specifically regulated by P/Q type Ca2+ channels at mammalian hippocampal synapses. Cell Reports 21, 341-350.

McGuinness L, Taylor C, Taylor RDT, Yau C, Langenhan T, Hart ML, Christian H , Tynan PW, Donnelly P, Emptage NJ (2010) Presynaptic NMDARs in the hippocampus facilitate transmitter release at theta frequency. Neuron 68, 1109-1127.

Ward B, McGunness L, Akerman C, Fine A, Bliss TVP, Emptage NJ (2006) State-dependent mechanisms of LTP expression revealed by optical quantal analysis. Neuron 52, 649-661.

Emptage NJ, Reid C, Fine A, Bliss TVP (2003) Optical quantal analysis reveals a presynaptic component of LTP at hippocampal Schaffer-associational synapses. Neuron 38, 797-804

Emptage NJ, Reid CA, Fine A (2001) Calcium stores in hippocampal synaptic boutons mediate short-term plasticity, store-operated Ca2+ entry, and spontaneous transmitter release. Neuron 29, 197-208.

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